Where I see patients (3)
Selected research
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LNK/SH2B3 as a novel driver in juvenile myelomonocytic leukemia.
Haematologica
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Allosteric SHP2 inhibition increases apoptotic dependency on BCL2 and synergizes with venetoclax in FLT3- and KIT-mutant AML.
Cell reports. Medicine
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The FLT3N701K mutation causes clinical AML resistance to gilteritinib and triggers TKI sensitivity switch to quizartinib.
American journal of hematology
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