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Home > Doctors > Saleh Adi

Saleh Adi, M.D.

Pediatric endocrinologist

Dr. Saleh Adi is a pediatric endocrinologist, diabetes specialist and director of Pediatric Diabetes Outpatient Services at UCSF Benioff Children's Hospital. He treats endocrine disorders including diabetes and adrenal, calcium, growth, pituitary, pubertal and thyroid disorders. He also conducts research on type 1 diabetes.

Adi is a member of the Endocrine Society, Lawson Wilkins Pediatric Endocrine Society and American Diabetes Association. He also is a member of the board of directors of the Juvenile Diabetes Research Foundation. His awards include the Clinical Investigator Award from the National Institutes of Health and the Solo Cup Clinician Scientist Award from the Johns Hopkins School of Medicine. He is an associate clinical professor of pediatrics at UCSF.

Clinics

Endocrinology Clinic
400 Parnassus Ave, Second floor
San Francisco, CA 94143
Phone: (415) 353-7337

Pediatric Diabetes Clinic
1500 Owens St., Suite 300
San Francisco, CA 94158
Phone: (415) 514-6234

Conditions & Treatments

More about Saleh Adi

Additional Languages

Arabic
French

Education

University of Aleppo School of Medicine, Syria 1984

Residencies

Veterans Administration Medical Center, Metabolics 1991
California Pacific Medical Center, Pediatrics 1994

Fellowships

UCSF Medical Center, Pediatric Endocrinology 1997

Selected Research and Publications

  1. Herold KC, Gitelman SE, Willi SM, Gottlieb PA, Waldron-Lynch F, Devine L, Sherr J, Rosenthal SM, Adi S, Jalaludin MY, Michels AW, Dziura J, Bluestone JA. Teplizumab treatment may improve C-peptide responses in participants with type 1 diabetes after the new-onset period: a randomised controlled trial. Diabetologia. 2013 Feb; 56(2):391-400.
  2. Adi S. Type 1 diabetes mellitus in adolescents. Adolesc Med State Art Rev. 2010 Apr; 21(1):86-102, ix.
  3. Tiffin N, Adi S, Stokoe D, Wu NY, Rosenthal SM. Akt phosphorylation is not sufficient for insulin-like growth factor-stimulated myogenin expression but must be accompanied by down-regulation of mitogen-activated protein kinase/extracellular signal-regulated kinase phosphorylation. Endocrinology. 2004 Nov; 145(11):4991-6.
  4. Dowell P, Otto TC, Adi S, Lane MD. Convergence of peroxisome proliferator-activated receptor gamma and Foxo1 signaling pathways. J Biol Chem. 2003 Nov 14; 278(46):45485-91.
  5. Adi S, Bin-Abbas B, Wu NY, Rosenthal SM. Early stimulation and late inhibition of extracellular signal-regulated kinase 1/2 phosphorylation by IGF-I: a potential mechanism mediating the switch in IGF-I action on skeletal muscle cell differentiation. Endocrinology. 2002 Feb; 143(2):511-6.
  6. Adi S, Wu NY, Rosenthal SM. Growth factor-stimulated phosphorylation of Akt and p70(S6K) is differentially inhibited by LY294002 and Wortmannin. Endocrinology. 2001 Jan; 142(1):498-501.
  7. Cheng ZQ, Adi S, Wu NY, Hsiao D, Woo EJ, Filvaroff EH, Gustafson TA, Rosenthal SM. Functional inactivation of the IGF-I receptor delays differentiation of skeletal muscle cells. J Endocrinol. 2000 Oct; 167(1):175-82.
  8. Adi S, Cheng ZQ, Zhang PL, Wu NY, Mellon SH, Rosenthal SM. Opposing early inhibitory and late stimulatory effects of insulin-like growth factor-I on myogenin gene transcription. J Cell Biochem. 2000 Jun 12; 78(4):617-26.
  9. Memon RA, Feingold KR, Moser AH, Doerrler W, Adi S, Dinarello CA, Grunfeld C. Differential effects of interleukin-1 and tumor necrosis factor on ketogenesis. Am J Physiol. 1992 Aug; 263(2 Pt 1):E301-9.
  10. Adi S, Pollock AS, Shigenaga JK, Moser AH, Feingold KR, Grunfeld C. Role for monokines in the metabolic effects of endotoxin. Interferon-gamma restores responsiveness of C3H/HeJ mice in vivo. J Clin Invest. 1992 May; 89(5):1603-9.
  11. Schwartz Z, Soskolne WA, Neubauer T, Goldstein M, Adi S, Ornoy A. Direct and sex-specific enhancement of bone formation and calcification by sex steroids in fetal mice long bone in vitro (biochemical and morphometric study. Endocrinology. 1991 Sep; 129(3):1167-74.
  12. Grunfeld C, Soued M, Adi S, Moser AH, Fiers W, Dinarello CA, Feingold KR. Interleukin 4 inhibits stimulation of hepatic lipogenesis by tumor necrosis factor, interleukin 1, and interleukin 6 but not by interferon-alpha. Cancer Res. 1991 Jun 1; 51(11):2803-7.
  13. Feingold KR, Soued M, Adi S, Staprans I, Neese R, Shigenaga J, Doerrler W, Moser A, Dinarello CA, Grunfeld C. Effect of interleukin-1 on lipid metabolism in the rat. Similarities to and differences from tumor necrosis factor. Arterioscler Thromb. 1991 May-Jun; 11(3):495-500.
  14. Feingold KR, Soued M, Adi S, Staprans I, Shigenaga J, Doerrler W, Moser A, Grunfeld C. Tumor necrosis factor-increased hepatic very-low-density lipoprotein production and increased serum triglyceride levels in diabetic rats. Diabetes. 1990 Dec; 39(12):1569-74.
  15. Feingold KR, Moser A, Adi S, Soued M, Grunfeld C. Small intestinal fatty acid synthesis is increased in diabetic rats. Endocrinology. 1990 Nov; 127(5):2247-52.
  16. Feingold KR, Adi S, Staprans I, Moser AH, Neese R, Verdier JA, Doerrler W, Grunfeld C. Diet affects the mechanisms by which TNF stimulates hepatic triglyceride production. Am J Physiol. 1990 Aug; 259(2 Pt 1):E177-84.
  17. Grunfeld C, Adi S, Soued M, Moser A, Fiers W, Feingold KR. Search for mediators of the lipogenic effects of tumor necrosis factor: potential role for interleukin 6. Cancer Res. 1990 Jul 15; 50(14):4233-8.
  18. Grunfeld C, Soued M, Adi S, Moser AH, Dinarello CA, Feingold KR. Evidence for two classes of cytokines that stimulate hepatic lipogenesis: relationships among tumor necrosis factor, interleukin-1 and interferon-alpha. Endocrinology. 1990 Jul; 127(1):46-54.
  19. Feingold KR, Soued M, Serio MK, Adi S, Moser AH, Grunfeld C. The effect of diet on tumor necrosis factor stimulation of hepatic lipogenesis. Metabolism. 1990 Jun; 39(6):623-32.
  20. Feingold KR, Serio MK, Adi S, Moser AH, Grunfeld C. Tumor necrosis factor stimulates hepatic lipid synthesis and secretion. Endocrinology. 1989 May; 124(5):2336-42.

Publications are derived from MEDLINE/PubMed and provided by UCSF Profiles, a service of the Clinical & Translational Science Institute (CTSI) at UCSF. Researchers can make corrections and additions by logging on to UCSF Profiles.

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